Metalloenzymes and Myocardial Infarction
Generate an AI Snapshot to get a quick, structured summary of this paper.
A concise AI-generated summary of the paper will appear here once you click Generate AI Snapshot.
TL;DR
Enzymatic and immunologic technics indicate the existence of a linear relation between the serum copper and ceruloplasmin and oxidase activity in pregnancy, infection and the nephrotic syndrome.
Abstract
ALTHOUGH 90 per cent or more of the copper content of normal human blood serum can be accounted for by the metalloenzyme, ceruloplasmin, few studies concerning the variability of this protein with changes in serum copper concentration are available. Variation of serum oxidase activity in proportion to the copper content has been shown in leukemia and polyarthritis and in the neonatal period.1 Enzymatic and immunologic technics indicate the existence of a linear relation between the serum copper and ceruloplasmin and oxidase activity in pregnancy, infection and the nephrotic syndrome.2 In Wilson's disease, a disproportionate decrease of serum copper concentration and . . .