The novel estrogenic receptor GPR30 alleviates ischemic injury by inhibiting TLR4-mediated microglial inflammation
Journal of NeuroinflammationPublished 12 July 2018Open access
Zengli Zhang, Pei Qin, Youliang Deng, Zhi Ma, Hang Guo, Haiyun Guo
Citations197
SJR quartileQ1
SJR score3.18
SNIP1.99
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TL;DR
Microglial G PR30 exerts acute neuroprotective effects by inhibiting TLR4-mediated microglial inflammation, which indicates that GPR30 may be a potential target for the treatment of ischemic stroke.
Abstract
Microglial GPR30 exerts acute neuroprotective effects by inhibiting TLR4-mediated microglial inflammation, which indicates that GPR30 may be a potential target for the treatment of ischemic stroke.
Keywords
MedicineNeuroscienceBiochemistry, Genetics and Molecular Biology
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