Cerebral metabolism during vegetative state and after recovery to consciousness
Journal of Neurology Neurosurgery & PsychiatryPublished 1 July 1999Open access
Steven Laureys, Christian Lemaire, Pierre Maquet, Christophe Phillips, G. Franck
Citations201
SJR quartileQ1
SJR score3.38
SNIP2.41
Generate an AI Snapshot to get a quick, structured summary of this paper.
Study Snapshot
ObjectiveStudy objective
MethodsResearch methodology
PopulationPopulation studied
Sample sizeSample sizes
OutcomesStudy outcomes here
ResultsStudy results comes here
LimitationsResearch study limitations comes here
A concise AI-generated summary of the paper will appear here once you click Generate AI Snapshot.
Abstract
peer reviewed
Keywords
Medicine
Human Brain Function
1,686 Citations1997R. S. J. Frackowiak
Principles and methods: Linking brain and behaviour, C. Frith Analyzing brain images - principles and overview, K. Friston Characterizing brain images with the general linear model, A. Holmes et al Making statistical inferences, J. Poline et al Characterizing distributed functional systems, K Friston characterizing functional integration.
New England Journal of MedicineMedical Aspects of the Persistent Vegetative State
1,520 Citations1994The Multi-Society Task Force on PVS
This consensus statement of the Multi-Society Task Force summarizes current knowledge of the medical aspects of the persistent vegetative state in adults and children.
Journal of Neuropathology & Experimental NeurologyNeuropathology of the Persistent Vegetative State. A Review
150 Citations1994Hannah C. Kinney, Martin A. Samuels
Archives of NeurologyBrain Glucose Metabolism in Postanoxic Syndrome
128 Citations1990Anne Devolder
The data suggest that brain anoxia can result in global brain hypometabolism, which appears related to the vigilance state, as well as in regional alterations preferentially located in arterial border zones.
PubMedAnti-ganglioside antibody and neuropathy: review of our research.
79 Citations1998Nobukazu Yuki
The molecular mimicry between infectious agents and gangliosides may function in the production of anti-ganglioside antibodies and the development of GBS and FS, and immunoadsorption with the column should be considered as an alternative form of plasmapheresis for the anti-GQ1b IgG antibody syndrome.
NeurologyTransient inhibitory seizures mimicking crescendo TIAs
67 Citations1990Henry Lee, Alfred M. Lerner
A patient with somatic inhibitory seizures who initially presented with a clinical picture of crescendo transient ischemic attacks does not improve with anticoagulation, but the episodes ceased promptly after the administration of an anticonvulsant.
Journal of Postgraduates of MedicineCAMPYLOBACTER JEJUNI INFECTION AND GUILLAIN-BARRSYNDROME
42 Citations2000Ma Jian
Cj infection is one of the important inducing factors in GBS; the presence of anti-GM1 antibody in the serum was associated with a recent Cj infection, but CJ infection or the presenceof anti- GM1 antibody was not associated with disease severity.
