Shear stress may stimulate release and action of nitric oxide in the human fetal-placental vasculature
American Journal of Obstetrics and GynecologyPublished 1 September 1995
Kathleen M. Wieczorek, Anthony S. Brewer, Leslie Myatt
Citations50
SJR quartileQ1
SJR score2.77
SNIP2.75
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TL;DR
Altering shear stress by increasing fetal flow rate or viscosity of the medium may increase synthesis and release of nitric oxide, which attenuates increases in perfusion pressure.
Abstract
Altering shear stress by increasing fetal flow rate or viscosity of the medium may increase synthesis and release of nitric oxide, which attenuates increases in perfusion pressure.
Keywords
Medicine
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